The authors reviewed the current literature regarding arthrogenic muscle and the  therapeutic interventions that may help clinicians.

AMI is caused by a change in the discharge of articular sensory receptors due to factors such as swelling, inflammation, joint laxity, and damage to joint afferents. Spinal reflex pathways that likely contribute to AMI include the group I nonreciprocal inhibitory pathway, the flexion reflex, and the gamma-loop. Preliminary evidence suggests that supraspinal pathways may also play an important role. 

Some of the most promising interventions to counter the effects of AMI include cryotherapy, transcutaneous electrical nerve stimulation, and neuromuscular electrical stimulation. Nonsteroidal anti-inflammatory drugs and intra-articular corticosteroids may also be effective when a strong inflammatory component is present with articular pathology.
 

AMI remains a significant barrier to effective rehabilitation in patients  following knee injury and surgery. Gaining a better understanding of AMI’s underlying mechanisms will allow the development of improved therapeutic strategies, enhancing the rehabilitationof patients with knee joint pathology.

Rice, D. A., & McNair, P. J. (2010). Quadriceps Arthrogenic Muscle Inhibition: Neural Mechanisms and Treatment Perspectives. Semin Arthritis Rheum, 40(3), 250–266.